While a multitude of reasons for these inconsistencies exist, controlling for dietary protein intake seems to be an area that previous research has not adequately considered. Results surrounding the ability of BCAAs to favorably impact recovery from damaging exercise are mixed. found that muscle soreness and MVIC in 26 recreationally active men were not affected by 0.1 g/kg of BCAA ingested pre- and post-damaging neuromuscular electrostimulation exercise. However, no differences in percent change for electrically stimulated maximal isometric force of the quadriceps, plasma CK, myoglobin and interleukin-6 response between groups post-exercise were observed. Jackman and investigators reported that compared to placebo treatment, 29.2 g of BCAA per day resulted in decreased DOMS at 48 and 72 hr in 24 non-resistance trained males after unilateral eccentric exercise. In addition, Sharp and colleagues supplemented eight recreationally active men with either a placebo or 6 g of BCAA for three weeks and reported a reduction in CK levels 12 and 36 hr after completing two days of intense resistance exercise. No differences were noted for vertical jump. In comparison to placebo, plasma creatine kinase (CK), perceived soreness and force production were all improved for the first 24 hr while soreness remained significantly lower up to 48 hr after damaging exercise in the BCAA supplemented group. examined the impact of 12 days of two daily doses of 10 grams (g) of BCAA or placebo in trained males who completed a workout consisting of 100 drop-jumps. While underlying mechanisms remain unclear, BCAA supplementation is a popular practice among recreational exercisers and athletes and continues to garner significant research interest. It has been suggested that BCAA supplementation may reduce protein degradation and/or muscle enzyme release, decrease skeletal muscle damage in response to intense resistance exercise, reduce feelings of soreness, mitigate central fatigue and promote subsequent recovery of muscle function however, these findings remain inconclusive at present. BCAAs (i.e., leucine, isoleucine and valine) are distinct among essential amino acids in that they are extrahepatically metabolized in skeletal muscle. Protein and amino acid supplements, including branched-chain amino acids (BCAA), have been considered a potentially efficacious dietary intervention. As such, nutritional strategies have been proposed to mitigate the negative effects that may be experienced following strenuous resistance exercise. The degree of damage and discomfort may be compounded over time and persist chronically, especially in individuals frequently engaging in vigorous exercise or those completing an overreaching phase. Skeletal muscle damage induced by resistance-based exercise is known to promote microdamage in muscle fibers, which may lead to temporary increased passive tension, delayed onset muscle soreness (DOMS), decrements in strength and force production, and increased efflux of intramuscular proteins into the blood. However, when consumed with a diet consisting of ~1.2 g/kg/day protein, the attenuation of muscular performance decrements or corresponding plasma CK levels are likely negligible. BCAA supplementation may mitigate muscle soreness following muscle-damaging exercise. No significant difference between groups ( p > 0.05) was detected for VJ or JS. CK concentrations were elevated above baseline ( p 0.05) at 24, 48 and 72 hr for BCAA individuals. No significant ( p > 0.05) group by time interaction effects were observed for CK, soreness, MVIC, VJ, or JS. Creatine kinase (CK), vertical jump (VJ), maximal voluntary isometric contraction (MVIC), jump squat (JS) and perceived soreness were assessed. Twenty males ingested either a BCAA supplement or placebo (PLCB) prior to and following eccentric exercise. This study investigated the effect of branched-chain amino acid (BCAA) supplementation on recovery from eccentric exercise.
0 kommentar(er)
